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By M. Pfreundschuh, H. Shiku, T. Takahashi, R. Ueda (auth.), Professor Georges Mathé, Professor Franco M. Muggia (eds.)

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Extra resources for Cancer Chemo- and Immunopharmacology: 2: Immunopharmacology, Relations, and General Problems

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J Immunol 116: 1251-1256 8 Kurland JT, Bockman R (1978) Prostaglandin E production by human blood monocytes and mouse peritoneal macrophages. J Exp Med 147: 952-957 9 Opitz HG, Niethammer D, Jackson RC, Lemke H, Huget R, Flad HD (1975) Biochemical characterization of a factor realised by macrophages. Cell Immunol 18: 70-75 10 Schechter GP, Soeh Len F (1978) Monocyte mediated inhibition of lymphocyte blastogenesis in Hodgkin's disease. Blood 52: 261-271 11 Sibbit WL, Bankhurst AD, Williams RC (1978) Studies of cell subpopulations mediating mitogen hyporesponsiveness in patients with Hodgkin's disease.

Malvine BAER provided editorial assistance. This work was supported by NIH contract NOI-CB-74185 21 Host's Immune State and Kinetics of Local Tumor Growth Control homologous tumor cells in parallel with the excision of the local tumor abrogated the enhancement of metastatic development in tumor-excised mice [7]. The accelerated growth of metastases was prevented also when local 3LL tumors of certain sizes were excised simultaneously with splenectomy [6]. In immunosuppressed animals, metastatic growth was enhanced, but an additional increase in incidence and growth rate of metastasis occurred after surgical removal of the local 3LL tumor [6].

In case a, in which the lymphocyte reactivity was practically abolished for both doses of PHA, lymphocyte responses recovered completely, reaching levels of reactivity from 60,000 to more than 100,000 cpm. In case b, in which only the response to PHA2 was depressed, a sixfold increase led to a response in the normal range. In this case, in which the response to PHA1 was normal, a 50% increase was observed, too. Finally, in case c, in which reactivity to PHA1 and to PHA2 was normal, the response improved, reaching a near twofold increase for the lymphocyte response to PHA2 .

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