By Hans-Joachim Priebe; Karl Skarvan
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Assessment of myocardial metabolism by PET: A sophisticated dream or clinical reality? Eur J Nucl Med 1986;12:570–5. 89 McMillin-Wood JB. Biochemical approaches in metabolism: application to positron emission tomography. Circulation 1985;72:IV145-50. 90 Taegtmeyer H, Mossberg KA, Nguyen VTB. Positron labelled tracers: A window for the assessment of energy metabolism in heart and skeletal muscle. Acta Radiol 1991;376:40–44. 91 Lewandowski ED. Nuclear magnetic resonance evaluation of metabolic and respiratory support of work load in intact rabbit hearts.
Science 1986;232:1121–3. 22 Kupriyanov VV, Lakomkin VL, Kapelko VI, Steinschneider AY, Ruuge EK, Saks VA. Dissociation of adenosine triphosphate levels and contractile function in isovolumic hearts perfused with 2-deoxyglucose. J Mol Cell Cardiol 1987;19:729–40. 23 Barth E, Stämmler G, Speiser B, Schaper J. Ultrastructural quantitation of mitochondria and myofilaments in cardiac muscle from 10 different animal species including man. J Mol Cell Cardiol 1992;24:669–81. 24 25 26 McNutt NS, Fawcett DW.
The fibre shortening (and ejection) is terminated when the maximal wall force that can be sustained by the myocardial fibres at the given level of contractility has been generated. This Page 31 force is defined by the systolic or active P/V relationship of the isovolumically contracting ventricle and is independent of preload. 1 5 The wall force during ejection is a function of ventricular size and geometry, and of developed pressure. As ventricular size decreases in the course of ejection, the instantaneous wall force and hence the ventricular afterload decrease.