By Iven Klineberg, Steven Eckert
Assess and deal with occlusion issues of confidence! practical Occlusion in Restorative Dentistry and Prosthodontics provides a full-color, accomplished consultant to occlusion, with assurance starting from an evidence of organic rules to therapy making plans and scientific tactics. An easy-to-understand method advances your talents with the newest evidence-based medical study, and reinforces wisdom with bankruptcy synopses, key issues packing containers, and plentiful references. To aid the book’s content material, a spouse site comprises 3 videos: condyle pursuits in the glenoid fossa, courting among condylar translation and rotation, and stream of the condyle–disc meeting recorded by means of MRI. Edited via across the world popular prosthodontics specialists Iven Klineberg and Steven Eckert, this reference might help you deal with occlusion stipulations and TMJ problems accurately and effectively.
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Extra resources for Functional Occlusion in Restorative Dentistry and Prosthodontics, 1e
Example text
Studies have shown that the extracellular matrix components of one periodontal compartment can influence the cellular activities of adjacent structures (Newman et al. 2012). Therefore the pathological changes occurring in one periodontal component may have significant ramifications for the maintenance, repair, or regeneration of other components of the periodontium (Newman et al. 2). Gingival Crevicular Fluid The gingival tissues and gingival crevicular fluid have been shown to contain a complex array of immune components that not only irrigate the gingival sulcus but also are released into the oral cavity (Ebersole 2003, Ebersole et al.
CD8 expressing T-cells). These T-cells are considered to be proactively maintaining tissue homeostasis in the face of the microbial challenge of the plaque biofilm (Gemmell et al. 2007; Newman et al. 2012). In contrast, in active periodontitis, B-cells and plasma cells predominate and are associated with pocket formation and 30 progression of disease. The periodontium is often compared to other mucosal tissues and the skin in terms of its repertoire of immune cells, and it contains a number of “professional” antigen-presenting cells (Cutler & Jotwani 2006; Cutler & Teng 2007).
OPG levels are regulated by transforming growth factor-β-related bone morphogenic proteins, whereas the synthesis 29 CHAPTER 2 | Periodontal Microbiology and Immunobiology of RANKL is induced by proinflammatory cyto kines such as IL-1β and TNF. Therefore increases in the concentration of proinflammatory cytokines in healthy periodontal tissue can directly affect bone loss by increasing the RANKL/OPG ratio (Belibasakis 2012, Darveau 2010). “Pathological” versus “Physiological” Bone Loss It is likely that the fundamental biological mechanisms associated with “pathological” bone loss as a result of periodontitis are similar to those associated with “physiological” bone loss from periodontal ligament widening associated with tooth mobility.