By Humberto Foyaca-Sibat (Ed.)
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Extra info for Novel Aspects on Epilepsy
Sample text
The complex has a stable interaction with stoichiometry of 1:1. The tight binding interaction between the two proteins formed a tumour suppressor heterodimer (Kwiatkowaki, 2008). Hamartin and tuberin have been found to physically associate with one another in vivo. Disruption in either one of the two genes may result in a truncated protein with the loss in controlling the cell growth and proliferation. The 130 kDa hamartin contains a putative transmembrane domain. , 1998). Although TSC1- or TSC2-specific functions are possible, it seems that the predominant biochemical activity of these proteins is exerted by an equimolar complex, which regulates the state of GTP-loading of the rheb GTPase, and thereby regulates mTOR activation in the cell.
The function loss can alternatively compromise neural migration via Tuberous Sclerosis Complex 21 interaction with ERM or actin binding proteins. It was also shown to activate the GTPase Rho and regulates focal adhesion and stress fiber formation. , 2000). , 2000). It has also been recently shown that the functional complex interact with G2/M cyclindependant kinase 1 and its regulatory cyclins. , 2001). The functional heteromeric complex of hamartin and tuberin also plays important role in modulating the pathways of insulin receptor-or insulin-like growth factor-mediated signalling.
It is recommended to avoid antiepileptic therapy with sedative effects (barbiturates, benzodiazepines) and useless combination of too many antiepileptic drugs. Correct sleep hygiene and life-style with regular and sufficient night-sleep and optimal surroundings is also important (Bazil, 2003, Happe, 2003). Hypersomnia in patients with epilepsy may be caused by sleep apnoe syndrome. This should be considered particulary in obese patients with morning headache and hypertension. Diagnosis is estimated by polysomnography.