By Ian Johnston
The situation identified most generally as pseudotumor cerebri syndrome is of diagnostic curiosity and scientific value not only to neurosurgeons, but in addition to neurologists, ophthalmologists and headache experts. Variously referred to as idiopathic intracranial high blood pressure, benign intracranial high blood pressure, and different names over the century or so because it was once first acknowledged, the authors argue for the grouping of a majority of these stipulations less than the identify of pseudotumor cerebri syndrome at the foundation of a standard underlying mechanism - an impairment of CSF absorption as a result of abnormalities on the CSF/venous interface. The ebook stories the advance of rules round a few of the extra contentious concerns and offers intensive with aetiology, investigative findings and techniques, therapy and final result, and within the concluding bankruptcy, considers the opportunity of setting up an experimental version to facilitate research of the unresolved matters, and pointing how to a extra whole knowing of this debatable situation.
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Additional resources for The Pseudotumor Cerebri Syndrome: Pseudotumor Cerebri, Idiopathic Intracranial Hypertension, Benign Intracranial Hypertension and Related Conditions
Sample text
Again, these authors postulated a dilatation of cerebral capacitance vessels to account for the increased CBV and also presumed that an increase in tissue volume due to oedema was the major contributing factor to the apparent fall in CBF. Reference was made to the CT study of Spallone (1981) to support this contention. They therefore concluded that raised ICP in PTCS was associated with a major dilatation of intraparenchymal cerebral vessels with increased CBV, but that this was unlikely to account for the rise in ICP.
1965) attributed the first clinical description of hypervitaminosis A to Joseph in 1944 and described five cases of their own, all children aged 1 to 6 months who had clinical evidence of raised ICP. Prior to that, Marie and See in 1954 described three cases, one with a measured pressure increase on lumbar puncture. Feldman and Schlezinger (1970) described two cases of a typical PTCS in adults associated with chronic hypovitaminosis A and, in a detailed review of the literature, identified six other cases in adults reported prior to 1970.
There are, however, three studies or groups of studies which pursue a particular line on mechanism and should be mentioned here, although they will be dealt with in greater detail in the following chapter. They are those of Reid et al. 26 History of the pseudotumor cerebri concept (1980, 1981) favouring brain oedema based on CT evidence of ventricular size, those of Sugerman et al. (1995, 1999) arguing for causative role for obesity, and those of King et al. (1995) and Karahalios et al. (1996) suggesting a much expanded causative role for increased cranial venous outflow pressure.