By Samuel Soskin (Eds.)
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In view of the known goiterogenic action of certain foods (see p. 29), the latter factor may have been of considerable importance. Even if it is granted that the administration of iodine was responsible for the differences in the incidence of goiter, it does 27 28 THYROID not follow that this reduction resulted from its making good a deficiency. It may have had a pharmacodynamic action. The small amounts of iodide used may seem to argue against this. It is true the amounts of iodide used for other purposes are much greater, but the concentration in the tissues to be affected may not be.
1* F O S T E R : 3rd Meeting in Miniature, Phila. Sec. Am. Chem. Soc. Catalyst 34: 21, 1949. ΐ δ M C C L E N D O N , J . F . : Letter to the editor. J . Clin. Endocrinol. 6: 589-590, 1946. l e M E R K E : 2nd Int. Goiter Conference, p. 254. Bern, Hans Huber, 1935. " L O V E , A. G . , A N D D A V E N P O R T , C . B . : Defects Found in Drafted Men. Washing ton, D . C , G O V . Pr. , 1919. 18 C A V E T T , J . W . , R I C E , C . , A N D M C C L E N D O N , J . F . : Thyroglobulin studies: The thyroxine and iodine content of normal and goitrous human thyroglobulin.
Nevertheless, the early statements relative to the malig nant degeneration of adenomas continue to be quoted. Some writers pre sent evidence of an etiologic relationship between nodular goiters generally and malignant tumors. Others believe that the danger of cancer arising on the basis of ordinary nodular goiter is relatively insignificant, and are of the opinion that it is the true benign adenoma which is the frequent pre cursor of carcinoma. Both of these propositions must be considered. While there is a distinct trend toward the differentiation of nodular goiter from true benign adenoma, there is no general agreement as to the existence, frequency, etiology, or clinical manifestations of true adenomas as contrasted with nodular goiter.